AFRICA CARDIOVASCULAR JOURNAL OF AFRICA • SMC-PAFCIC Abstracts October 2022 44 (1.2 - 10.627), p=0.021). D-dimer was not detected as a risk factor, and this can be explained by the characteristics of our population. Although the previous use of anti-thrombotic drugs protects against thrombo-embolic complications during severe infection, there was no benefit in terms of mortality (Figure2). Conclusion: Prior use of antithrombotic drugs is a protective factor against thromboembolic complications in patients with a history of heart disease but has no effect on mortality. Figure 1 Figure 2 Submission ID: 1595 TOO MUCH SUGAR BLOCKS YOUR HEART? SINOATRIAL ARRESTS ASSOCIATED WITH DIABETIC KETOACIDOSIS REGRESSING TO SINUS RHYTHM AFTER COMPENSATION OF DIABETIC PROCESS ABDERRAHMANE BOUCHAALA, JAOUAD NGUADI, NAJAT MOUINE, HICHAM BOUZELMAT, AATIF BENYASS MOROCCO Background: Ketoacidosis is a serious life-threatening condition due to its profound hydro electrolytic disturbances andmultiple complications. Ketoacidosis itself is induced by numerous conditions and diseases, of which diabetic decompensation is one of the frequent etiologies. We report the case of a patient presenting to the emergency department for diabetic ketoacidosis decompensation, complicated by multiple sinoatrial arrests regressing after medical treatment. A review of the literature will be discussed in order to expose the different possible etiopathogenic mechanisms. Case presentation: A 51-years-old man with 7-year history of type 2 diabetes under oral anti-diabetic drugs, two previous hospitalizations for ketoacidosis decompensations, triggered by a pulmonary infection and poor therapeutic compliance respectively, presented to the emergency department for sluggishness, asthenia associated with diffuse abdominal pain. Clinical examination found a somnolent patient, polypneic at 27 cpmwith correct roomair saturation, his blood pressure figures were 97/61 mmHg and bradycardia at 37 bpm. Abdominal examination found diffuse tenderness without specific localization of pain or hepatosplenomegaly, the hernial orifices were free. The initial blood analysis showed hyperglycemia at 6,1 g/L, acetone and glycosuria were detectable with urine dipsticks, in addition a blood gas analysis revealed a metabolic acidosis with high anion gap (Tab. 1). The diagnosis of ketoacidosis decompensation was established with medical treatment and search for triggering factors started. The initial ECG showed the presence of numerous sinoatrial pauses and arrests of variable duration (between 2 and 8 seconds) with idioventricular escape (Fig.1). Figure 1 Table 1: Biological parameters of the patient on admission to the emergency department. Figure 2: Return to sinus rhythm. MODERATED POSTER SESSION
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