Cardiovascular Journal of Africa: Vol 33 No 5 (SEPTEMBER/OCTOBER 2022)

CARDIOVASCULAR JOURNAL OF AFRICA • Volume 33, No 5, September/October 2022 242 AFRICA Arterioscler Thromb Vasc Biol 2004; 24(12): 2414–2419. 60. Vaziri ND. Molecular mechanisms of lipid disorders in nephrotic syndrome. Kidney Int 2003; 63(5): 1964–1976. 61. Pai JK, Pischon T, Ma J, Manson JE, Hankinson SE, Joshipura K, et al. Inflammatory markers and the risk of coronary heart disease in men and women. N Engl J Med 2004; 351(25): 2599–2610. 62. Stehouwer CD, Fischer HA, Van Kuijk AW, Polak BC, Donker AJ. Endothelial dysfunction precedes development of microalbuminuria in IDDM. Diabetes 1995; 44(5): 561–564. 63. Scurt FG, Menne J, Brandt S, Bernhardt A, Mertens PR, Haller H, et al. Systemic inflammation precedes microalbuminuria in diabetes. Kidney Int Rep 2019; 4(10): 1373–1386. 64. Von Scholten BJ, Reinhard H, Hansen TW, Schalkwijk CG, Stehouwer C, Parving H-H, et al. Markers of inflammation and endothelial dysfunction are associated with incident cardiovascular disease, all-cause mortality, and progression of coronary calcification in type 2 diabetic patients with microalbuminuria. J Diabetes Complicat 2016; 30(2): 248–255. 65. Rubio-Guerra AF, Vargas-Robles H, Lozano Nuevo JJ, EscalanteAcosta BA. Correlation between Circulating adhesion molecule levels and albuminuria in type-2 diabetic hypertensive patients. Kidney Blood Press Res 2009; 32(2): 106–109. 66. Ezzat EM, Razek GA, Zaki O. Microalbuminuria as a marker of cardiovascular risks in Egyptian hypertensive patients. Asian J Med Principles Clin Prac 2018: 1–5. 67. Moriwaki Y, Yamamoto T, Shibutani Y, Aoki E, Tsutsumi Z, Takahashi S, et al. Elevated levels of interleukin-18 and tumor necrosis factor-α in serum of patients with type 2 diabetes mellitus: relationship with diabetic nephropathy. Metabolism 2003; 52(5): 605–608. 68. Rodrigues M, Dias CB. Microalbuminuria in non diabetic population as an marker of nephropathy. Brazilian J Nephrol 2016; 38(2): 203–208. 69. Ozkurt ZN, Ebinc FA, KeleŞ H, Ebinc H, Güliter S. Frequency of microalbuminuria and its relationship with other atherosclerotic risk factors in nondiabetic hypertensive patients. Anadolu Kardiyol Derg 2007; 7(2): 224–226. 70. Tseng C-H. Differential dyslipidemia associated with albuminuria in type 2 diabetic patients in Taiwan. Clin Biochem 2009; 42(10–11): 1019–1024. 71. Cook I. Objectively-measured physical activity patterns and longitudinal weight category status in a rural setting. BMC Res Notes 2019; 12(1): 624. 72. Cook I. Do low levels of physical activity in female adolescents cause overweight and obesity? Objectively measured physical activity levels of periurban and rural adolescents. S Afr Med J 2015; 105(8): 659–663. 73. Astor BC, Matsushita K, Gansevoort RT, van der Velde M, Woodward M, Levey AS, et al. 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Hydration may slow cardiac decline and reduce heart failure risk: NIH study Staying well-hydrated may slow down the decline in cardiac function and decrease the prevalence of heart failure, according to researchers at the National Institutes of Health (NIH), reported in the European Heart Journal. While fluid guidelines vary based on the body’s needs, the researchers recommended a daily fluid intake of six to eight cups (1.5– 2.1 litres) for women and eight to 12 cups (2–3 litres) for men. Heart failure, a chronic condition that develops when the heart does not pump enough blood for the body’s needs, affects more than 6.2 million Americans, a little more than 2% of the population. It is also more common among adults aged 65 years and older. ‘Similar to reducing salt intake, drinking enough water and staying hydrated are ways to support our hearts and may help reduce long-term risks for heart disease,’ said Natalia Dmitrieva, PhD, the lead study author and a researcher in the Laboratory of Cardiovascular Regenerative Medicine at the National Heart, Lung, and Blood Institute (NHLBI), part of NIH. After conducting pre-clinical research that suggested connections between dehydration and cardiac fibrosis, a hardening of the heart muscles, Dmitrieva and researchers looked for similar associations in large-scale population studies. To start, they analysed data from more than 15 000 adults, aged 45–66 years, who enrolled in the Atherosclerosis Risk in Communities (ARIC) study between 1987 and 1989 and shared information from medical visits over a 25-year period. In selecting participants for their retrospective review, the scientists focused on those whose hydration levels were within a normal range and who did not have diabetes, obesity or heart failure at the start of the study. Approximately 11 814 adults were included in the final analysis, and, of those, the researchers found that 1 366 (11.56%) later developed heart failure. To assess potential links with hydration, the team assessed the hydration status of the participants, using several clinical measures. Looking at levels of serum sodium, which increases as the body’s fluid levels decrease, was especially useful in helping to identify participants with an increased risk for developing heart failure. It also helped identify older adults with an increased risk for developing both heart failure and left ventricular hypertrophy, an enlargement and thickening of the heart. continued on page 259…

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